AMP-Activated Protein Kinase (AMPK) and Estrogen-Dependent Mechanisms Underlying Increased Susceptibility to Cardiovascular Disease during Menopause

Text Registration

AMP-Activated Protein Kinase (AMPK) and Estrogen-Dependent Mechanisms Underlying Increased Susceptibility to Cardiovascular Disease during Menopause

Status

Published

on 1 Jun 2020

Year of Creation

2020

Marissa Pier

Registration Number

TX0008879411

on 1 Jun 2020

The U.S. Copyright record (Registration Number: TX0008879411) dated 1 Jun 2020, pertains to an electronic file (eService) titled "AMP-Activated Protein Kinase (AMPK) and Estrogen-Dependent Mechanisms Underlying Increased Susceptibility to Cardiovascular Disease during Menopause" created in 2020. The copyright holder is Marissa Pier, known for their creative contributions in text registration. For any inquiries concerning this copyrighted material, kindly reach out to Marissa Pier.

Marissa Pier

Application Details

Registration Number

TX0008879411

Registration Date

6/1/2020

Year of Creation

2020

Agency Marc Code

DLC-CO

Record Status

New

Physical Description

Electronic file (eService)

First Publication Nation

United States

Personal Authors

Marissa Pier

Notes

Rights Note: Mark Dill, ProQuest, LLC, 789 E. Eisenhower Parkway, Ann Arbor, MI, 48108-3218, United States, (800) 521-0600, disspub@proquest.com

Statements

Application Title Statement: AMP-Activated Protein Kinase (AMPK) and Estrogen-Dependent Mechanisms Underlying Increased Susceptibility to Cardiovascular Disease during Menopause

Author Statement: Marissa Pier Citizenship: not known Authorship: text

Notes

Rights Note: Mark Dill, ProQuest, LLC, 789 E. Eisenhower Parkway, Ann Arbor, MI, 48108-3218, United States, (800) 521-0600, disspub@proquest.com

Statements

Application Title Statement: AMP-Activated Protein Kinase (AMPK) and Estrogen-Dependent Mechanisms Underlying Increased Susceptibility to Cardiovascular Disease during Menopause

Author Statement: Marissa Pier Citizenship: not known Authorship: text

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